Trigeminal neuralgia is an alteration of 5th cranial nerve. Let's see below natural cures for trigeminal neuralgia, its causes and official medical



It causes crises of intense pain in face, forehead, teeth and internal mucous membranes of the jaw and jowl. It has a prevalence in women (male /

female ratio of 1: 1.8) and affects 2.5% to 5.7% of people. Multiple sclerosis is present in 2-4% of patients with trigeminal neuralgia, often the

younger ones.


Cause of Trigeminal Neuralgia is damage to the myelin sheath (lining) of this nerve. This makes electrical impulses traveling through it erratic or

excessive, activating algogenic regions (where pain starts) in brain. The damage can be caused by a vascular-nervous conflict due to an artery

with abnormal course, which compresses nerve. The superior cerebellar artery is often responsible (with the continuous pulsations that cause

microtrauma to myelin sheath).


But biological cause of trigeminal inflammation is explained by dysregulation of mast cell metabolism. This dysregulation causes an excessive and

chaotic introduction into local and systemic blood circulation of neural mediators (VEGF-vascular endothelial growth factors). They cause an

increasingly severe inflammatory state of trigeminal, already damaged by compression or by systemic diseases (multiple sclerosis). But, natural

cures for trigeminal neuralgia are able to dominate dysregulation of mast cells without harm the body.


Episodes of pain occur suddenly, triggered by common activities or exposure to cold. Patients report feeling like intermittent electric shocks. Single

attacks affect one side of face at a time, last for a few seconds, and can come and go throughout the day (even for several months). In 3-5% of cases

disease is bilateral and attacks can increase in frequency or severity over time. Many patients describe areas on face that trigger pain (trigger points).

That's, a simple touch or even air flow can trigger a painful episode.


Current treatment of trigeminal neuralgia can be pharmacological and / or surgical. Drugs considered in treatment of trigeminal neuralgia are

anticonvulsant: carbamazepine or gabapentin. If, as in half of cases, drugs are not tolerated or not effective, surgical treatment should be used.

This therapy consists of some procedures through the skin (trigeminal radiofrequency thermorizotomy, selective neurolysis). Surgery (microvascular

decompression which involves craniotomy) can also be performed. These methods can have serious complications. Furthermore, they do not solve

problem as they have a high relapse rate (from 20% to 80% at 5 and 10 years). As a consequence of these procedings, an atypical trigeminal

neuralgia (sub-continuous pain which may be accompanied by paresthesia, burning) may also occur.


Radiosurgical treatment (Gamma-Knife or Cyber-Knife) consists of administering a high dose of rays (X or gamma) to emergence of 5th cranial

nerve. Sensitive fibers (more fragile) thus lose their functionality while motor fibers usually do not report any damage. However, with this method

there is a high percentage of facial disorders. In fact, pain can turn into an unpleasant sensation of tingling or burning, often more annoying than

trigeminal neuralgia itself. And later on, it's no longer possible to perform other procedures, having now caused nerve damage.


So far the official solutions. Instead, let's consider concrete possibility of a natural cure for trigeminal neuralgia. It does'nt involve any side

effects or contraindications and effectively solve the problem. By acting in order to restore normal functioning of nerve fiber lining cells. In fact,

it's possible to restore mast cells metabolism with phytotherapeutic substances like Rhodiola rosea and Vitis vinifera. They have an anti-free

radical, anti-inflammatory, anti-aggregating and nourishing action on blood vessel wall. Then, with the help of other natural substances, such as

flavoproteins and coenzymes, ther's an harmless way of rebalancing cell membranes. The production of neurotransmitters (VEGF) returns to

normal. All this avoids nerve damage and promotes the recovery of neuronal function.